Pharmacologic blockade of MCH-R1 receptor reduces cataplexy and REM sleep in narcoleptic mice

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منابع مشابه

Systemic administration of hypocretin-1 reduces cataplexy and normalizes sleep and waking durations in narcoleptic dogs.

Recent work has implicated the hypocretin (orexin) system in the genesis of narcolepsy. In the current study we demonstrate that systemically administered hypocretin-1 (Hcrt-1) produces an increase in activity level, longer waking periods, a decrease in REM sleep without change in nonREM sleep, reduced sleep fragmentation and a dose dependent reduction in cataplexy in canine narcoleptics. Repea...

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STUDY OBJECTIVES We recently proposed insufficient non-rapid eye movement sleep (NREMS) intensity to contribute to disturbed nocturnal sleep in patients with narcolepsy-cataplexy (NC). To test this hypothesis, we investigated the effect of physiologically intensified NREMS in recovery sleep following sleep deprivation. DESIGN Nocturnal baseline and recovery sleep architecture, and the sleep e...

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Melanin-Concentrating Hormone (MCH): Role in REM Sleep and Depression

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REM sleep respiratory behaviours mental content in narcoleptic lucid dreamers

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Narcolepsy has been hypothesized to be a disease of rapid eye movement (REM) sleep. According to this hypothesis, cataplexy is a result of the triggering during waking of the mechanism that normally serves to suppress muscle tone in REM sleep. REM sleep control mechanisms have been localized to the pons. Narcoleptic dogs have increased numbers of cholinergic receptors in the medial pons. These ...

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ژورنال

عنوان ژورنال: Frontiers in Neuroscience

سال: 2011

ISSN: 1662-453X

DOI: 10.3389/conf.fnins.2011.84.00143